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LETTER TO EDITOR |
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| Year : 2008 | Volume
: 56
| Issue : 1 | Page : 90-91 |
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Spontaneous late-onset comitant acute nonaccommodative esotropia in children
Uday V Naik, Sumita Agarkar, Meenakshi Swaminathan, TS Surendran
Pediatric Ophthalmology Department, Sankara Nethralaya, 18, College Road, Chennai - 600 006, India
Correspondence Address:
Uday V Naik
Pediatric Ophthalmology Department, Sankara Nethralaya, 18, College Road, Chennai - 600 006
India
DOI: 10.4103/0301-4738.37612 PMID: 18158425
How to cite this article:
Naik UV, Agarkar S, Swaminathan M, Surendran T S. Spontaneous late-onset comitant acute nonaccommodative esotropia in children. Indian J Ophthalmol 2008;56:90-1 |
How to cite this URL:
Naik UV, Agarkar S, Swaminathan M, Surendran T S. Spontaneous late-onset comitant acute nonaccommodative esotropia in children. Indian J Ophthalmol [serial online] 2008 [cited 2013 Jun 19];56:90-1. Available from: http://www.ijo.in/text.asp?2008/56/1/90/37612 |
Dear Editor,
We read the article by Kothari,[1] with great interest. The author has described the clinical characteristics of spontaneous, late-onset comitant acute nonaccommodative esotropia in children. We appreciate that the author has drawn attention to this relatively uncommon clinical entity. We would like to put forward a different view regarding neuroimaging in such cases.
Many intracranial lesions present with comitant nonaccommodative esotropia without neurological deficit in early stage.[2] Sometimes acute-onset nonaccommodative esotropia is the only presenting sign of intracranial neoplasm. There are many studies in the literature which state that intracranial lesions may present with acute-onset comitant esotropia without any neurological deficit or other signs. [2],[3],[4]
In patients with acute esotropia with corpus callosum gliomas, clinical signs are absent in the early part of the disease. Also, tumors involving the corpus callosum give rise to no distinctive signs.[3] Posterior fossa lesions are typically known to present with only acute-onset comitant esotropia with no other systemic signs such as papilledema.[2]
Acute comitant esotropia has also been described in patients with Arnold Chiari malformation More Details even prior to onset of typical down beating nystagmus.[4] It is important in these cases of acquired esotropia to rule out the possibility of intracranial tumor, Arnold Chiari malformation or other neurological abnormalities.
Although most children with this form of esotropia are otherwise healthy, central nervous system lesions must be considred.[5]
Missing a brain tumor, however rare, in a child presenting with acquired comitant nonaccommodative esotropia will be an act of negligence. So we feel that neuroimaging should be done in any patient presenting with acquired comitant nonaccommodative esotropia.
 References | |  |
| 1. | Kothari M. Clinical characteristics of spontaneous late-onset comitant acute nonaccommodative esotropia in children. Indian J Ophthalmol 2007;55:117-20.  |
| 2. | Williams AS, Hoyt CS. Acute comitant esotropia in children with brain tumors. Arch Ophthalmol 1989;107:376-8. [PUBMED] |
| 3. | Anderson W, Lubon M. Astrocytoma of the corpus callosum presenting with acute comitant esotropia. Am J Ophthalmol 1970;69:594-8. |
| 4. | Bixeman MW, Laguna JF. Acquired esotropia as initial manifestation of Arnold-Chiari malformation. J Pediatr Ophthalmol Strabismus 1987;24:83-6. |
| 5. | Pediatric Ophthalmology and Strabismus, AAO, BCSC, Chapter 7, page no 104. |
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