Home About us Editorial board Ahead of print Current issue Search Archives Submit article Instructions Subscribe Contacts Login 
  • Users Online: 3228
  • Home
  • Print this page
  • Email this page

   Table of Contents      
LETTER TO THE EDITOR
Year : 2008  |  Volume : 56  |  Issue : 4  |  Page : 343-344

Authors' reply


Aravind Eye Hospital and Postgraduate Institute of Ophthalmology, Madurai, Tamil Nadu, India

Date of Web Publication19-Jun-2008

Correspondence Address:
Vasumathy Vedantham
Consultant, Retina - Vitreous Service, Aravind Eye Hospital and Postgraduate Institute of Ophthalmology, 1, Anna Nagar, Madurai - 625 020, Tamil Nadu
India
Login to access the Email id

Source of Support: None, Conflict of Interest: None


DOI: 10.4103/0301-4738.41429

Rights and Permissions

How to cite this article:
Vedantham V, Jethani J, Agarwal A, Vijayalakshmi P. Authors' reply. Indian J Ophthalmol 2008;56:343-4

How to cite this URL:
Vedantham V, Jethani J, Agarwal A, Vijayalakshmi P. Authors' reply. Indian J Ophthalmol [serial online] 2008 [cited 2020 Apr 7];56:343-4. Available from: http://www.ijo.in/text.asp?2008/56/4/343/41429

Dear Editor,

We thank the reader [1] for his interest in our article [2] while we also approach his concerns that inferior oblique overaction can be because of superior oblique dysfunction. However, the very term primary inferior oblique overaction implies that such a possibility has already been excluded. Needless to say our patient underwent a detailed orthoptic workup including measurement of deviation in nine gazes and either head tilt that together with evaluation of duction did not suggest any extraocular muscle dysfunction. The same could not be included in the article because of restrictions in word limit.

We are surprised that the reader should link blepharophimosis to mesodermal dysgenesis and dysfunction of extraocular muscles, no such association has been reported to the best of our knowledge. Further, the eyelids are ectodermally derived and we recommend the reader to refer the book, [3] which states that the entity of blepharophimosis syndrome is predominantly a dysplasia of the eyelids consisting of inverted inner canthal fold, short palpebral fissure with lateral displacement of inner canthi, low nasal bridge, and ptosis of eyelids with hypoplasia and fibrosis of levator palpebrae superioris.

 
  References Top

1.
Pandey PK, Vats P, Kaur N, Kulkarni AG. Primary inferior oblique over action as part of a new syndrome. Indian J Ophthalmol 2008;56:343-4.  Back to cited text no. 1
[PUBMED]  Medknow Journal  
2.
Vedantham V, Jethani J, Agarwal A, Vijayalakshmi P. Retinitis pigmentosa associated with blepharophimosis, blue dot cataract and primary inferior oblique overaction: A new syndrome complex?. Indian J Ophthalmol 2007;55:150-1  Back to cited text no. 2
    
3.
Smith's recognizable pattern of Human Malformation, Blepharophimosis syndrome. Philadelphia: W.B. Saunders Co; p. 194-5.  Back to cited text no. 3
    




 

Top
 
 
  Search
 
    Similar in PUBMED
   Search Pubmed for
   Search in Google Scholar for
    Access Statistics
    Email Alert *
    Add to My List *
* Registration required (free)  

 
  In this article
References

 Article Access Statistics
    Viewed1214    
    Printed25    
    Emailed0    
    PDF Downloaded89    
    Comments [Add]    

Recommend this journal