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   Table of Contents      
LETTER TO THE EDITOR
Year : 2013  |  Volume : 61  |  Issue : 9  |  Page : 533-534

Incidence of pupillary involvement, course of anisocoria and ophthalmoplegia in diabetic Oculomotor nerve palsy


Department of General Opthalmology, Aravind Eye Hospital, Thavalakuppam, Cuddalore, Pondicherry, India

Date of Web Publication8-Oct-2013

Correspondence Address:
Sahil Bhandari
Aravind Eye Hospital, Thavalakuppam, Cuddalore Main Road, Pondicherry - 605 007
India
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Source of Support: None, Conflict of Interest: None


DOI: 10.4103/0301-4738.119462

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How to cite this article:
Bhandari S, Yadalla D. Incidence of pupillary involvement, course of anisocoria and ophthalmoplegia in diabetic Oculomotor nerve palsy. Indian J Ophthalmol 2013;61:533-4

How to cite this URL:
Bhandari S, Yadalla D. Incidence of pupillary involvement, course of anisocoria and ophthalmoplegia in diabetic Oculomotor nerve palsy. Indian J Ophthalmol [serial online] 2013 [cited 2019 Dec 9];61:533-4. Available from: http://www.ijo.in/text.asp?2013/61/9/533/119462

Dear Editor,

After going through the article published in the January 2013 issue on "Incidence of pupillary involvement, course of anisocoria and ophthalmoplegia in diabetic Oculomotor nerve palsy" I have few queries:

  1. Cases recruited in the study had multiple systemic diseases other than diabetes, which can also cause ischemic Oculomotor palsy independently. So how was it decided that only diabetes is the cause of Oculomotor palsy? [1]
  2. Was there any co-relation between the status of systemic diseases at presentation and follow-up, and course of anisocoria and ophthalmoplegia? If no, then can the pupillary involvement be attributed only to diabetes? [2],[3]
  3. If the study was not a masked trial, then there could have been subjective variation in measuring the pupil size and reaction, which can alter the final inference.
  4. Does the absence of diabetic retinopathy, not point towards other cause of nerve palsy, as the pathogenesis of both is considered to be microangiopathy. [4],[5]
I would also like to suggest that near vision, near point of accommodation of the involved eye could have been taken into account for analyzing the course of pupillary involvement.

Appreciable fact about the study is that if there is anisocoria of <2 mm in nerve palsy cases then we need not subject the patient to costly Neuro-imaging.

 
  References Top

1.
Dhume KU, Paul KE. Incidence of pupillary involvement, course of anisocoria and ophthalmoplegia in diabetic oculomotor nerve palsy. Indian J Ophthalmol 2013;61:13-7.  Back to cited text no. 1
[PUBMED]  Medknow Journal  
2.
Walsh and Hoyt's Clinical Neuro-Ophthalmology, 6 th ed, vol 1. Section III; The Ocular Motor System; 20-Nuclear and Infranuclear Ocular MotilityDisorders.  Back to cited text no. 2
    
3.
Boschi A. Neuro-ophthalmological problems in the diabetic patient. Bull Soc Belge Opthamol 1995;256:145-50.  Back to cited text no. 3
    
4.
Akagi T, Miyamoto K, Kashii S, Yoshimura N. Cause and prognosis of neurologically isolated third, fourth, or sixth cranial nerve dysfunction in cases of oculomotor palsy. Jpn J Ophthalmol 2008;52:32-5.  Back to cited text no. 4
    
5.
Greco D, Gambina F, Maggio F. Ophthalmoplegia in diabetes mellitus: A retrospective study. Acta Diabetol 2009;46:23-6.  Back to cited text no. 5
    




 

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