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Year : 1983  |  Volume : 31  |  Issue : 7  |  Page : 1025-1026

Pan retinal photo coagulation in diabetic retinopathy

Institute of Ophthalmology & Sarojini Devi Eye Hospital, Hyderabad, India

Correspondence Address:
M Satpathi
Institute of Ophthalmology & Sarojini Devi Eye Hospital, Hyderabad, A.P.
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Source of Support: None, Conflict of Interest: None

PMID: 6544248

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How to cite this article:
Satpathi M. Pan retinal photo coagulation in diabetic retinopathy. Indian J Ophthalmol 1983;31, Suppl S1:1025-6

How to cite this URL:
Satpathi M. Pan retinal photo coagulation in diabetic retinopathy. Indian J Ophthalmol [serial online] 1983 [cited 2021 Sep 26];31, Suppl S1:1025-6. Available from: https://www.ijo.in/text.asp?1983/31/7/1025/29736

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Table 1

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Table 1

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Diabetes with retinopathy develop pro­liferative changes in 5% of 10%. These occur in the presence of severe microvascular abnor­lities, particularly capillary non perfusion which results in retinal ischemia. It is pos­tulated that vaso-formation substances are released which stimulates the growth of the new vessels. These usually occur at the edge of the areas of the capillary non perfusion in the peripheral retina and at the optic disc. New vessels leak plasma components and may bleed resulting in pre-retinal and vitreous haemorrhage. The vitreous body contracts away from the retinal surface pulling the fragile new vessels with it. The mechanical traction on the blood vessels increases the ten­dency to haemorrhage and formation of fib­rous band results in detachment of the retina.

Early treatment of all the areas of retinal ischemic by peripheral retinal ablation pre­vents blindness in the majority of cases. The aim of the treatment is to protect the foveal vision by destroying all the peripheral retina as far as the equator. Areas of ischemic retina at the posterior pole, as noted by nonperfu­sion of capillaries on fluorascein angior­gaphy must be destroyed.

  Material and methods Top

Those patients on routine screening to have diabetic retinopathy were referred from eye specialists and diabetic clinics are selected for this study.

Patients entering the record were subjected to full ophthalmic assessment including ref­raction, tonometry. Visual fields, fundus photography and fluorescein angiography. Every attempt was made to achieve a good diabetic controll.

The Criteria for inclusion were that 1) the patient affected with proliferative diabetic retinopathy process involving the retina and optic disc. 2) that the visual acuity at initial assessment is not less than 6/24 in the pro­posed eye for treatment.

Exclusion of cases from the study was made 1) advanced stage of proliferative retinopathy 2) Visual acuity was adversely effected by opacities of the media 3) Cases with ne­phropathy and extra ocular complications 4) in cases of Maculopathy.

  Technique Top

All treated eyes were subjected to peri­pheral retinal photocoagulation by means of a Log 3 Xenon light coagulator. The pro­cedure was well tolerated after instillation of local anesthetic drops and retrobulbar xylocaine.

Photocoagulation burns were applied within the vascular arcades above and below the macula but an approximately circular area of ardius 2½ disc diameters, with the fovea at its centre and including the papi lomacular bundle. In the nasal fundus photocoagulation was extended centrally to the edge of the optic disc and involved any new vessels arising from the margin.

Individual burns are made with an image field aperture 2° to 3° (.6 mm to 0.9 m.m.) and an exposure time 1 second so that uniform grey colour is seen over the treated ares. A white coagulam is considered as exessive energy to complete the treatment over 300 burns in 6 sittings in a period of 3 weeks to the most of our patients. During the subsequent visits, photo-coagulation was applied to cover up the ara left in earlier sittings.

Patients were reviewed at an itervals of 1 month and 6 months after cmpletion of peripheral retinal ablation.

  Observation and conclusion Top

More number of cases are presented with peripheral retinal Neo-vascularisation.

Evaluation of treatment was done basing on four main criteria. 1. Visual acuity. 2. Appearanc of Optic disc and peripheral retina often 6 months. 3. Any other compli­cations.

Visual acutiy was maintained as before and there was no deteroration of vision in 46 (60%) of the cases and there was regression of teh neo vascularisation botn in the optic disc and over the peripheral retina.

2. There was no complication encoun­tered during photocoagulation.

3. 20 (25%) cases have developed fresh vitreo-retinal haemorrhage during the follow up of 2 years.

4. 12 (15%) of the cases have developed optic atirophy and deterioration of vision from the level during the follow up period.[1]

  References Top

Constable San J. 1981, Monogram. Laser its clinical uses in eye diseases.  Back to cited text no. 1


  [Table - 1], [Table - 2], [Table - 3], [Table - 4]


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